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• W2033766281 abstract "We used cultured neonatal rat cardiac myocytes to test the hypothesis that all-trans retinoic acid (atRA) may act to modulate ANG II actions in inducing myocyte hypertrophy. Our observations were as follows. 1) atRA (10(-7) to approximately 10(-5) M ) inhibited ANG II-induced hyperplasia of fibroblasts in a dose-dependent manner. 2) Treatment of atRA attenuated the ANG II-induced increase in total cell protein content. 3) Treated with ANG II (10(-7) M) for 5 days, the cultured neonatal rat cardiac myocytes demonstrated an apparent accumulation of sarcomeric fiber proteins and Golgi's complex, as well as reorganization of the sarcomeric unit within individual myocytes. atRA (10(-6) M) treatment reduced the accumulation of contractile proteins and Golgi's complex without affecting the ANG II-induced reorganization of the sarcomeric unit. 4) atRA attenuated the ANG II-induced increase in intracellular Ca2+. Our results show that atRA inhibits some effects of ANG II on neonatal rat cardiac myocytes and suggest that atRA may be a therapeutic candidate for the prevention and therapy of cardiac hypertrophy and remodeling." @default.
• W2033766281 created "2016-06-24" @default.
• W2033766281 creator A5066504632 @default.
• W2033766281 creator A5076130185 @default.
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• W2033766281 date "2002-05-01" @default.
• W2033766281 modified "2023-10-17" @default.
• W2033766281 title "Effects of<i>all</i>-<i>trans</i>retinoic acid on angiotensin II-induced myocyte hypertrophy" @default.
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• W2033766281 doi "https://doi.org/10.1152/japplphysiol.01192.2001" @default.
• W2033766281 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/11960970" @default.
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