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• W2033965329 abstract "1 The effects of bethanechol and atropine on the release of acetylcholine (ACh) from bullfrog sympathetic preganglionic nerve terminals were examined electrophysiologically. 2 Bethanechol (1 mM) caused no depolarization of sympathetic preganglionic nerve terminals, whereas carbachol or ACh in the same concentration induced marked depolarizations of these terminals. 3 Bethanechol (10 microM) depressed the amplitude of fast excitatory postsynaptic potentials (e.p.s.ps) recorded in Ca2+-high Mg2+ solution, without depolarizing ganglion cells. The quantal content measured from these fast e.p.s.ps by the variance method showed a significant reduction. 4 Amplitudes of both miniature e.p.s.ps and ACh-potentials induced by iontophoresis of ACh were not affected by addition of bethanechol (10 microM). 5 The depressant effect of bethanechol (10 microM) on fast e.ps.ps disappeared in the presence of atropine (3 microM). 6 Atropine (3 microM) increased the quantal content measured from fast e.p.s.ps recorded in low Ca2+-high Mg2+ solution. 7 The depressant effect of bethanechol (10 microM) on fast e.p.s.ps was unaffected by alpha-adrenoceptor blocking agents (phenoxybenzamine (10 microM) or phentolamine (10 microM). 8 These results suggest that presynaptic nerve terminals in bullfrog sympathetic ganglia possess a muscarinic receptor which inhibits active release of ACh." @default.
• W2033965329 created "2016-06-24" @default.
• W2033965329 creator A5052239224 @default.
• W2033965329 creator A5059995923 @default.
• W2033965329 date "1982-09-01" @default.
• W2033965329 modified "2023-09-26" @default.
• W2033965329 title "PRESYNAPTIC MUSCARINIC RECEPTORS INHIBITING ACTIVE ACETYLCHOLINE RELEASE IN THE BULLFROG SYMPATHETIC GANGLION" @default.
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• W2033965329 doi "https://doi.org/10.1111/j.1476-5381.1982.tb09271.x" @default.
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