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• W2033990421 abstract "Epidemiological studies have consistently shown that moderate alcohol consumption is associated with a reduced risk of acute myocardial infarction (AMI) [ 1 Yano K. Rhoads G.G. Kagan A. Coffee, alcohol and risk of coronary heart disease among Japanese men living in Hawaii. N Engl J Med. 1977; 297: 405-409 Crossref PubMed Scopus (421) Google Scholar , 2 Klatsky A.L. Friedman G.D. Siegelaub A.B. Alcohol and mortality. A ten-year Kaiser-Permanente experience. Ann Intern Med. 1981; 95: 139-145 Crossref PubMed Scopus (326) Google Scholar , 3 Gordon T. Doyle J.T. Drinking and coronary heart disease: the Albany Study. Am Heart J. 1985; 110: 331-334 Abstract Full Text PDF PubMed Scopus (26) Google Scholar , 4 Miller G.J. Beckles G.L. Maude G.H. Carson D.C. Alcohol consumption: protection against coronary heart disease and risks to health. Int J Epidemiol. 1990; 19: 923-930 Crossref PubMed Scopus (55) Google Scholar , 5 Shaper A.G. Wannamethee G. Walker M. Alcohol and coronary heart disease: a perspective from the British Regional Heart Study. Int J Epidemiol. 1994; 23: 482-494 Crossref PubMed Scopus (74) Google Scholar , 6 Gaziano J.M. Buring J.E. Breslow J.L. et al. Moderate alcohol intake, increased levels of high-density lipoprotein and its subfractions, and decreased risk of myocardial infarction. N Engl J Med. 1993; 329: 1829-1834 Crossref PubMed Scopus (936) Google Scholar , 7 Kitamura A. Iso H. Sankai T. et al. Alcohol intake and premature coronary heart disease in urban Japanese men. Am J Epidemiol. 1998; 147: 59-65 Crossref PubMed Scopus (78) Google Scholar ]. Nearly half of the northern Asian population lacks the enzyme, aldehyde dehydrogenase 2 (ALDH2), that quickly metabolizes acetaldehyde (a metabolite of ethanol by alcohol dehydrogenase) into acetate, resulting in high acetaldehyde concentrations after drinking, as well as flushing and other symptoms [ 8 Takeshita T. Morimoto K. Mao X.-Q. Hashimoto T. Furuyama J. Phenotypic differences in low Km aldehyde dehydrogenase in Japanese workers. Lancet. 1993; 341: 837 Abstract PubMed Scopus (71) Google Scholar , 9 Harada S. Agarwal D.P. Goedde H.W. Aldehyde dehydrogenase deficiency as cause of facial flushing reaction to alcohol in Japanese. Lancet. 1981; 2: 982 Abstract PubMed Scopus (346) Google Scholar ]. A case–control study in Japan suggested that those who had alcohol-induced flushing had an increased risk of AMI [ [10] Takagi S. Iwai N. Yamauchi R. et al. Aldehyde dehydrogenase 2 gene is a risk factor for myocardial infarction in Japanese men. Hypertens Res. 2002; 25: 677-681 Crossref PubMed Scopus (129) Google Scholar ]. However, the number of subjects in this study was relatively small. Thus, we examined the association of alcohol consumption and incidence of AMI among middle-aged Japanese men with and without alcohol-induced flushing using data from a large cohort study." @default.
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• W2033990421 date "2007-10-01" @default.
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• W2033990421 title "Alcohol consumption, alcohol-induced flushing and incidence of acute myocardial infarction among middle-aged men in Japan—Japan Public Health Center-based prospective study" @default.
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• W2033990421 doi "https://doi.org/10.1016/j.atherosclerosis.2007.01.030" @default.
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