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- W2034116639 endingPage "1034" @default.
- W2034116639 startingPage "1025" @default.
- W2034116639 abstract "Smoking is a major health problem and is propelled, at least in part, by the addictive properties of nicotine. Two types of pharmacological therapies have been approved for smoking cessation by the US Food and Drug Administration. The first therapy consists of nicotine replacement, substituting the nicotine from cigarettes with safer nicotine formulations. The second therapy is bupropion (Zyban), an atypical antidepressant, whose use has raised much debate as to how a non-nicotine-based agent can aid in smoking cessation. This review focuses on recent advances that could lead to the development of improved novel pharmacological treatments. These strategies focus on altering reward processes in the brain by modulating various neurotransmitter systems: the most promising include dopamine D(3) receptor antagonists, noradrenaline reuptake inhibitors, GABA(B) receptor agonists, metabotropic glutamate 5 (mGluR5) receptor antagonists, cannabinoid CB1 receptor antagonists, and corticotropin releasing factor (CRF) 1 receptor antagonists." @default.
- W2034116639 created "2016-06-24" @default.
- W2034116639 creator A5032192926 @default.
- W2034116639 creator A5045075812 @default.
- W2034116639 creator A5073852920 @default.
- W2034116639 creator A5084928512 @default.
- W2034116639 date "2003-11-01" @default.
- W2034116639 modified "2023-10-16" @default.
- W2034116639 title "Non-nicotinic neuropharmacological strategies for nicotine dependence: beyond bupropion" @default.
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- W2034116639 doi "https://doi.org/10.1016/s1359-6446(03)02890-3" @default.
- W2034116639 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/14690633" @default.
- W2034116639 hasPublicationYear "2003" @default.
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