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- W2034149599 abstract "Intrastriatal administration of ammonium ions (ammonia) via a microdialysis probe overactivates N-methyl-D-aspartate (NMDA) receptors, which results in cGMP accumulation in the microdialysates. Co-administration of a potent glycine site-specific NMDA receptor antagonist CGP 78608 ([(1S)-1-[[(7-bromo-1,2,3,4-tetrahydro-2,3-dioxo-5-quinoxalinyl)methyl]amino]ethyl]phosphonate) significantly reduced (at 20 nM) or abolished (at 100 nM) ammonia-dependent cGMP synthesis. Since NMDA receptor activation is an important causative factor in ammonia neurotoxicity, the present results suggest the glycine site of the receptor to be a potential valuable target for protective intervention." @default.
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- W2034149599 date "2004-07-01" @default.
- W2034149599 modified "2023-10-16" @default.
- W2034149599 title "A novel glycine site-specific N-methyl-d-aspartate receptor antagonist prevents activation of the NMDA/NO/CGMP pathway by ammonia" @default.
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- W2034149599 doi "https://doi.org/10.1016/j.brainres.2004.05.014" @default.
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