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- W2034161006 endingPage "349" @default.
- W2034161006 startingPage "339" @default.
- W2034161006 abstract "Abstract As background for an antibody‐based therapeutic program against the IGF receptor, we undertook a review of available information on the early pregnancy‐specific regulation and localization of IGFs, IGF‐binding proteins (BPs), IGFBP‐specific proteases, and the type 1 IGF receptor relative to placental maintenance, function of placental nutrient transporters, placental cellular differentiation/turnover/apoptosis, and critical hormone signaling needed to maintain pregnancy. Possible adverse outcomes of altered IGF signaling include prenatal loss, fetal growth retardation, and maldevelopment are also discussed. It appears that the IGF axes in both the conceptus and mother are important for normal embryo‐fetal growth. Thus, all molecules (i.e., both small and large) that disrupt the IGF axis could be expected to have some degree of fetal consequences. Birth Defects Res (Part B) 89:339–349, 2010. © 2010 Wiley‐Liss, Inc." @default.
- W2034161006 created "2016-06-24" @default.
- W2034161006 creator A5035046865 @default.
- W2034161006 creator A5051692915 @default.
- W2034161006 creator A5053891807 @default.
- W2034161006 date "2010-06-10" @default.
- W2034161006 modified "2023-10-06" @default.
- W2034161006 title "Maternal‐placental insulin‐like growth factor (IGF) signaling and its importance to normal embryo‐fetal development" @default.
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