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• W2034171109 abstract "Significance The mechanisms that mediate bone morphogenetic protein (BMP) receptor recycling, and the importance of such recycling for signaling in vivo, have remained poorly understood. We find that the retromer complex functions as a linchpin in the recycling of the BMP type I receptor SMA-6 (small-6). In the absence of retromer-dependent recycling, retromer mutants result in the missorting of SMA-6 to lysosomes and a loss of BMP-mediated signaling. Surprisingly, we find that the BMP type II receptor, DAF-4 (dauer formation-defective-4), recycles through a distinct recycling pathway. Taken together, our results indicate a mechanism that separates the type I and type II receptors during receptor recycling, potentially terminating signaling while preserving both receptors for further rounds of activation." @default.
• W2034171109 created "2016-06-24" @default.
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• W2034171109 date "2014-02-03" @default.
• W2034171109 modified "2023-10-11" @default.
• W2034171109 title "BMP signaling requires retromer-dependent recycling of the type I receptor" @default.
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• W2034171109 doi "https://doi.org/10.1073/pnas.1319947111" @default.
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