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- W2034254855 endingPage "17" @default.
- W2034254855 startingPage "7" @default.
- W2034254855 abstract "Neuroinflammation is a pathological hallmark in Parkinson's disease (PD) and amyotrophic lateral sclerosis (ALS), and is characterized by activated microglia and infiltrating T cells at sites of neuronal injury. In PD and ALS, neurons do not die alone; neuronal injury is non-cell-autonomous and depends on a well-orchestrated dialogue in which neuronally secreted misfolded proteins activate microglia and initiate a self-propagating cycle of neurotoxicity. Diverse populations and phenotypes of CD4(+) T cells crosstalk with microglia, and depending on their activation status, influence this dialogue and promote neuroprotection or neurotoxicity. A greater understanding of the T cell population that mediates these effects, as well as the molecular signals involved should provide targets for neuroprotective immunomodulation to treat these devastating neurodegenerative diseases." @default.
- W2034254855 created "2016-06-24" @default.
- W2034254855 creator A5015630437 @default.
- W2034254855 creator A5032862233 @default.
- W2034254855 creator A5048445973 @default.
- W2034254855 date "2010-01-01" @default.
- W2034254855 modified "2023-09-30" @default.
- W2034254855 title "T cell-microglial dialogue in Parkinson's disease and amyotrophic lateral sclerosis: are we listening?" @default.
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