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• W2034268003 abstract "Catheter-based renal artery ablation selectively reduces both renal sympathetic efferent and afferent nerve activity and causes renal sympathetic denervation (RSD) [ [1] Krum H. Sobotka P. Mahfoud F. Böhm M. Esler M. Schlaich M. Device-based antihypertensive therapy: therapeutic modulation of the autonomic nervous system. Circulation. 2011; 123: 209-215 Crossref PubMed Scopus (132) Google Scholar ]. Some clinical trials and animal experiments have suggested that RSD could decrease the RAAS activity [ [2] Zhao Q. Yu S. Zou M. Effect of renal sympathetic denervation on the inducibility of atrial fibrillation during rapid atrial pacing. J Interv Card Electrophysiol. 2012; 35: 119-125 Crossref PubMed Scopus (52) Google Scholar ], and further has great potential in the treatment of heart failure (HF) [ [3] Davies J.E. Manisty C.H. Petraco R. et al. First-in-man safety evaluation of renal denervation for chronic systolic heart failure: Primary outcome from REACH-Pilot study. Int J Cardiol. 2013; 162: 189-192 Abstract Full Text Full Text PDF PubMed Scopus (227) Google Scholar ]. Whether RSD can attenuate ventricular arrhythmia (VA) and ventricular substrate remodeling induced by catheter-based renal artery ablation is unclear. Therefore, the purpose of the present study was to determine whether RSD can attenuate the inducibility of VA in dogs with pacing-induced HF." @default.
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• W2034268003 date "2014-07-01" @default.
• W2034268003 modified "2023-10-16" @default.
• W2034268003 title "Renal sympathetic denervation attenuates the ventricular substrate and electrophysiological remodeling in dogs with pacing-induced heart failure" @default.
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• W2034268003 doi "https://doi.org/10.1016/j.ijcard.2014.04.189" @default.
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