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- W2034336869 abstract "Cancer cells have been characterized with activated mutant oncogenes and inactivated or deleted tumor suppressor genes. Cancer cells are also aneuploid, displaying a jumble of chromosomal anomalies including gain or loss of whole chromosomes or transposed chromosomal fragments. Whether mutation of specific genes or aneuploidy is more critical for tumorigenesis is very much a contentious issue. We recently showed that activated oncogenes induce oxidative damage that is exacerbated by conventional cell culture conditions. This culture shock or a loss of p53 function creates a precarious environment that permits oncogenes to induce rapid chromosomal instability and transformation. We found that mutant genes and aneuploidy were prerequisites and collaborators for neoplastic transformation." @default.
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- W2034336869 date "2004-09-09" @default.
- W2034336869 modified "2023-10-14" @default.
- W2034336869 title "Gene Mutations and Aneuploidy: The Instability That Causes Cancer" @default.
- W2034336869 doi "https://doi.org/10.4161/cc.3.9.1089" @default.
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