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- W2034366080 abstract "<h3>To the Editor.</h3> —We read with great interest the article by Drs Allen and Epstein<sup>1</sup>demonstrating the additive effect of betaxolol and epinephrine in primary open angle glaucoma. They found that epinephrine increased outflow facility in the presence of betaxolol, but not in the presence of timolol. This led them to hypothesize that epinephrine increases outflow through a β<sub>2</sub>-adrenergic mechanism. We have recently demonstrated for the first time the presence of β-adrenergic receptors in human trabecular meshwork and in cultured trabecular cells.<sup>2</sup>Furthermore, we determined that the vast majority of the β-adrenergic receptors were of the β<sub>2</sub>-adrenergic subtype. Our findings were subsequently confirmed by Kurtz et al<sup>3</sup>using cultured human trabecular cells. These studies provide an anatomical basis for the concept that epinephrine acts through β<sub>2</sub>-adrenergic receptors in the trabecular meshwork to increase aqueous outflow facility." @default.
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- W2034366080 date "1987-01-01" @default.
- W2034366080 modified "2023-10-18" @default.
- W2034366080 title " -Adrenergic Receptors in Human Trabecular Meshwork" @default.
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- W2034366080 doi "https://doi.org/10.1001/archopht.1987.01060010024007" @default.
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