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- W2034380587 abstract "Senescence-accelerated mice (SAM), a murine model of age-related deterioration in learning ability, were studied as to the acetylcholine (ACh) contents in the brain tissues and the effect of nicotine administration. We found that the ACh content of SAM-P/8 (accelerated senescence-prone) mice was lower than that of SAM-R/1 (accelerated senescence-resistant) mice in the midbrain thalamus and the hypothalamus. In addition, an IP administration of nicotine was found to improve learning ability of SAM-P/8 as shown by performance of a passive avoidance task. Nicotine may potentiate cognitive function in SAM-P/8." @default.
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- W2034380587 date "1994-11-01" @default.
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- W2034380587 title "Nicotine imptoves cognitive disturbance in senescence-accelerated mice" @default.
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- W2034380587 doi "https://doi.org/10.1016/0091-3057(94)90100-7" @default.
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