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• W2034456143 abstract "Premenopausal females have a comparably lower incidence of cardiovascular disease than their male counterparts. Although estrogen and activation of estrogen receptors (ERs) have been found to contribute to female protection, the complex mechanisms involved are unclear. Besides altering gene transcription, estrogen could elicit its cardioprotective effect via ER-mediated nongenomic signaling pathways. In addition to the two classic nuclear ER isoforms, ERα and ERβ, a G-protein coupled ER (GPR30 or GPER) has been found to be expressed in cardiomyocytes and plays an acute cardioprotective role in ischemia reperfusion injury. By using isoform-specific ER knockout mouse models and/or their specific modulators, the mechanisms of the different ERs involved in cardioprotection have been explored. In this review, we will focus on the signaling pathways leading to cardioprotection in ischemia reperfusion injury after ER activation and discuss the possibility and promise of specific ER modulators to treat ischemic heart diseases." @default.
• W2034456143 created "2016-06-24" @default.
• W2034456143 creator A5064257229 @default.
• W2034456143 creator A5082173766 @default.
• W2034456143 creator A5089124744 @default.
• W2034456143 date "2010-04-01" @default.
• W2034456143 modified "2023-10-02" @default.
• W2034456143 title "Estrogen Receptor Activation and Cardioprotection in Ischemia Reperfusion Injury" @default.
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• W2034456143 doi "https://doi.org/10.1016/j.tcm.2010.05.001" @default.
• W2034456143 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/3011947" @default.
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