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• W2034602671 endingPage "576" @default.
• W2034602671 startingPage "568" @default.
• W2034602671 abstract "To establish axonal connections growth cones must navigate multiple intermediate targets before reaching their final target. During this journey growth cones are guided by extracellular repulsive and attractive signals. Although initially identified as repulsive molecules, members of the semaphorin family include both attractants and repellents. How a navigating growth cone responds to a specific semaphorin is not absolute but instead depends on the biological context in which this cue is encountered. Here we review recent breakthroughs in our understanding of the extrinsic signals and molecular processes that control growth cone responses to class 3 semaphorins (Sema3s) at a well-characterized intermediate target, the spinal cord midline. To establish axonal connections growth cones must navigate multiple intermediate targets before reaching their final target. During this journey growth cones are guided by extracellular repulsive and attractive signals. Although initially identified as repulsive molecules, members of the semaphorin family include both attractants and repellents. How a navigating growth cone responds to a specific semaphorin is not absolute but instead depends on the biological context in which this cue is encountered. Here we review recent breakthroughs in our understanding of the extrinsic signals and molecular processes that control growth cone responses to class 3 semaphorins (Sema3s) at a well-characterized intermediate target, the spinal cord midline." @default.
• W2034602671 created "2016-06-24" @default.
• W2034602671 creator A5021053685 @default.
• W2034602671 creator A5049167080 @default.
• W2034602671 creator A5064421447 @default.
• W2034602671 date "2010-09-01" @default.
• W2034602671 modified "2023-09-26" @default.
• W2034602671 title "Semaphorin signaling: molecular switches at the midline" @default.
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• W2034602671 doi "https://doi.org/10.1016/j.tcb.2010.06.007" @default.
• W2034602671 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/20655749" @default.
• W2034602671 hasPublicationYear "2010" @default.
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