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- W2034705919 abstract "Ischemia-reperfusion injury occurred in the kidneys in the cases of renal transplantation and hypovolemic shock causes severe inflammation and tissue damage of the kidneys, resulting in the acute rejection and renal failure. Recently, apoptosis following the inflammation has been reported to play a key role in the tissue damage. Inhibition of Fas/FasL-mediated signal which plays a central role in the apoptosis effectively protected the tissue from ischemia-reperfusion injury. Fas-Fc, ZVAD-fmk, Fas siRNA, and anti-FasL antibody significantly decreased the number of apoptotic cells, attenuated the tissue injury and reduced mortality. Because some of these agents were effective even they were given after ischemia-reperfusion, they may be potent agents to ameliorate the apoptosis-induced renal tissue injury." @default.
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- W2034705919 date "2007-01-01" @default.
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- W2034705919 title "Protection of renal tissue from ischemia-reperfusion injury by inhibition of Fas-induced apoptosis" @default.
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- W2034705919 doi "https://doi.org/10.2492/inflammregen.27.124" @default.
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