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• W2034773089 abstract "Type 1 diabetes (T1D) develops as a consequence of abnormal responses against several self-antigens, eventually leading to the autoimmune attack and destruction of the insulin-producing beta cells in the pancreas. In this issue of Laboratory Investigation, Li et al propose the transcription factor Pancreatic and duodenal homeobox 1 (PDX-1) as a T1D autoantigen by demonstrating autoreactivity to this pancreas-specific protein in both the NOD mouse model and patients with T1D. Because of the known roles of PDX-1 in pancreatic development as well as beta cell maintenance and function, targeting of PDX-1 expressing cells may result in the elimination of not only beta cells but also the progenitor cells required for regeneration of insulin-producing cells." @default.
• W2034773089 created "2016-06-24" @default.
• W2034773089 creator A5065752077 @default.
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• W2034773089 date "2010-01-01" @default.
• W2034773089 modified "2023-10-16" @default.
• W2034773089 title "Attacking the source: anti-PDX-1 responses in type 1 diabetes" @default.
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• W2034773089 doi "https://doi.org/10.1038/labinvest.2009.121" @default.
• W2034773089 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/3756500" @default.
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