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- W2034780161 abstract "Posttetanic potentiation (PTP) is an essential aspect of synaptic transmission that arises from a persistent presynaptic [Ca2+]i following tetanic stimulation. At crayfish neuromuscular junctions, several inhibitors of mitochondrial Ca2+ uptake and release (tetraphenylphosphonium or TPP+, carbonyl cyanide m-chlorophenylhydrazone or CCCP, and ruthenium red) blocked PTP and the persistence of presynaptic residual [Ca2+]i, while endoplasmic reticulum (ER) Ca2+ pump inhibitors and release channel activators (thapsigargin, 2,5-di-(tert-butyl)-1,4-benzohydroquinone or BHQ, and caffeine) had no effects. PTP apparently results from the slow efflux of tetanically accumulated mitochondrial Ca2+." @default.
- W2034780161 created "2016-06-24" @default.
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- W2034780161 date "1997-03-01" @default.
- W2034780161 modified "2023-10-17" @default.
- W2034780161 title "Mitochondrial Involvement in Post-Tetanic Potentiation of Synaptic Transmission" @default.
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- W2034780161 doi "https://doi.org/10.1016/s0896-6273(00)81248-9" @default.
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