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- W2034791666 abstract "The goal of this review is to provide an update on the different forms of monogenic diabetes, including maturity-onset diabetes of the young (MODY) and neonatal diabetes (permanent and transient neonatal diabetes).Monogenic diabetes accounts for approximately 1-2% of diabetes cases and results from mutations that primarily reduce β-cell function. Individuals with islet autoantibody negative youth-onset forms of diabetes should be evaluated for either glucokinase-MODY or transcription factors MODY. The mild-fasting hyperglycemia found in glucokinase-MODY typically does not necessitate pharmacological treatment, whereas patients with MODY caused by transcription factor mutations can often be successfully treated with low-dose sulfonylurea. Neonatal diabetes is defined as diabetes onset within the first 6 months of life and most individuals with permanent neonatal diabetes can be treated with high-dose sulfonylurea.The discovery of the genetic cause of monogenic diabetes has greatly advanced our understanding and management of these uncommon forms of diabetes." @default.
- W2034791666 created "2016-06-24" @default.
- W2034791666 creator A5006967177 @default.
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- W2034791666 date "2011-08-01" @default.
- W2034791666 modified "2023-09-26" @default.
- W2034791666 title "Review on monogenic diabetes" @default.
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- W2034791666 doi "https://doi.org/10.1097/med.0b013e3283488275" @default.
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