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- W2034823787 abstract "New findings on the role of LfR (lactotransferrin receptor), MTf (melanotransferrin), CP (ceruloplasmin) and DCT1 (Divalent Cation Transporter) in brain iron transport, obtained during the past 3 years, are important advances in the fields of physiology and pathophysiology of brain iron metabolism. According to these findings, disruption in the expression of these proteins in the brain is probably one of the important causes of the altered brain iron metabolism in age-related neurodegenerative diseases, including Parkinson's Disease, Alzheimer's disease, Huntington's disease and amyotrophic lateral sclerosis. Further studies on the involvement of LfR, MTf and DCT1 in iron uptake by and CP in iron egress from different types of brain cells as well as control mechanisms of expression of these proteins in the brain are critical for elucidating the causes of excessive accumulation of iron in the brain and neuronal death in neurodegenerative diseases." @default.
- W2034823787 created "2016-06-24" @default.
- W2034823787 creator A5031516698 @default.
- W2034823787 creator A5065243448 @default.
- W2034823787 date "1998-07-01" @default.
- W2034823787 modified "2023-10-14" @default.
- W2034823787 title "Expression of iron transport proteins and excessive iron accumulation in the brain in neurodegenerative disorders" @default.
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- W2034823787 doi "https://doi.org/10.1016/s0165-0173(98)00012-5" @default.
- W2034823787 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/9729418" @default.
- W2034823787 hasPublicationYear "1998" @default.
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