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• W2034864533 abstract "The development of obesity is the consequence of a multitude of complex interactions between both genetic and environmental factors. It has been suggested that the dramatic increase in the prevalence of obesity over the past 30 years has been the result of environmental changes that have enabled the full realization of genetic susceptibility present in the population. Among the many environmental alterations that have occurred in our recent history is the ever-increasing dyssynchrony between natural cycles of light/dark and altered patterns of sleep/wake and eating behavior associated with our “24-hour” lifestyle. An extensive research literature has established clear links between increased risk for obesity and both sleep deprivation and shift work, and our understanding of the consequences of such dyssynchrony at the molecular level is beginning to emerge. Studies linking alterations in cellular circadian clocks to metabolic dysfunction point to the increasing importance of chronobiology in obesity etiology." @default.
• W2034864533 created "2016-06-24" @default.
• W2034864533 creator A5001920865 @default.
• W2034864533 creator A5057371321 @default.
• W2034864533 date "2012-02-17" @default.
• W2034864533 modified "2023-10-14" @default.
• W2034864533 title "Chronobiological Effects on Obesity" @default.
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• W2034864533 doi "https://doi.org/10.1007/s13679-011-0005-4" @default.
• W2034864533 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/3653336" @default.
• W2034864533 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/23682347" @default.
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