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• W2035376077 abstract "During H5N1 influenza virus infection, proinflammatory cytokines are markedly elevated in the lungs of infected hosts. The significance of this dysregulated cytokine response in H5N1-mediated pathogenesis remains to be determined. To investigate the influence of hypercytokinemia, or cytokine storm, a transgenic mouse technology was used. The classical NF-kappaB pathway regulates the induction of most proinflammatory cytokines. Deletion of the p50 subunit leads to a markedly reduced expression of the NF-kappaB-regulated cytokines and chemokines. Here we show that H5N1 influenza virus infection of this transgenic mouse model resulted in a lack of hypercytokinemia but not in altered pathogenesis." @default.
• W2035376077 created "2016-06-24" @default.
• W2035376077 creator A5024773634 @default.
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• W2035376077 date "2008-11-15" @default.
• W2035376077 modified "2023-10-01" @default.
• W2035376077 title "Role of Hypercytokinemia in NF-κB p50-Deficient Mice after H5N1 Influenza A Virus Infection" @default.
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• W2035376077 doi "https://doi.org/10.1128/jvi.01071-08" @default.
• W2035376077 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/2573282" @default.
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