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- W2035503887 endingPage "404" @default.
- W2035503887 startingPage "385" @default.
- W2035503887 abstract "In rheumatoid arthritis (RA), cells within the inflamed synovium and pannus elaborate a variety of cytokines, including tumor necrosis factor (TNF) alpha, interleukin (IL)-1, IL-6, and IL-17, that contribute to inflammation, and may directly affect bone. The receptor activator of NF-kappaB (RANK) ligand/RANK/osteoprotegerin pathway plays a critical role in regulating osteoclastogenesis in articular bone erosions in RA. Proinflammatory cytokines can modulate this pathway, and may also affect the ability of the osteoblast to repair bone at sites of articular erosion. In this review, the authors discuss the current understanding of pathogenic mechanisms of bone erosion in RA and examine current therapeutic approaches to prevent this damage." @default.
- W2035503887 created "2016-06-24" @default.
- W2035503887 creator A5024038808 @default.
- W2035503887 creator A5028063859 @default.
- W2035503887 creator A5041049194 @default.
- W2035503887 date "2010-05-01" @default.
- W2035503887 modified "2023-10-04" @default.
- W2035503887 title "Bone Damage in Rheumatoid Arthritis: Mechanistic Insights and Approaches to Prevention" @default.
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