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• W2035531975 startingPage "392" @default.
• W2035531975 abstract "Endothelial cells express two classical cadherins, VE-cadherin and N-cadherin. VE-cadherin is absolutely required for vascular morphogenesis, but N-cadherin is thought to participate in vessel stabilization by interacting with periendothelial cells during vessel formation. However, recent data suggest a more critical role for N-cadherin in endothelium that would regulate angiogenesis, in part by controlling VE-cadherin expression. In this study, we have assessed N-cadherin function in vascular development using an in vitro model derived from embryonic stem (ES) cell differentiation. We show that pluripotent ES cells genetically null for N-cadherin can differentiate normally into endothelial cells. In addition, sprouting angiogenesis was unaltered, suggesting that N-cadherin is not essential for the early events of angiogenesis. However, the lack of N-cadherin led to an impairment in pericyte covering of endothelial outgrowths. We conclude that N-cadherin is necessary neither for vasculogenesis nor proliferation and migration of endothelial cells but is required for the subsequent maturation of endothelial sprouts by interacting with pericytes." @default.
• W2035531975 created "2016-06-24" @default.
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• W2035531975 creator A5090485594 @default.
• W2035531975 date "2005-11-01" @default.
• W2035531975 modified "2023-09-25" @default.
• W2035531975 title "N-cadherin deficiency impairs pericyte recruitment, and not endothelial differentiation or sprouting, in embryonic stem cell-derived angiogenesis" @default.
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• W2035531975 doi "https://doi.org/10.1016/j.yexcr.2005.08.021" @default.
• W2035531975 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/16202998" @default.
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