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- W2036385196 endingPage "67" @default.
- W2036385196 startingPage "63" @default.
- W2036385196 abstract "Normal cells have limited proliferative potential in culture, a fact that has been the basis of their use as a model for replicative senescence for many years. Recent molecular analyses have identified numerous changes in gene expression that occur as cells become senescent, and the results indicate that multiple levels of control contribute to the irreversible growth arrest. These include repression of growth stimulatory genes, overexpression of growth inhibitory genes, and interference with downstream pathways. Studies with cell types other than fibroblasts will better define the role of cell senescence in the aging process and in tumorigenesis." @default.
- W2036385196 created "2016-06-24" @default.
- W2036385196 creator A5024822764 @default.
- W2036385196 creator A5029881104 @default.
- W2036385196 date "1996-07-05" @default.
- W2036385196 modified "2023-10-18" @default.
- W2036385196 title "Replicative Senescence: Implications for in Vivo Aging and Tumor Suppression" @default.
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