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• W2036632501 abstract "The main purpose of the present study was to determine if high concentrations of oxygen (O2) would inhibit cell division in alveoli undergoing reparative regeneration following injury by ozone (O3). Mice were exposed to 2.5 ppm O3 for 6 hr to injure the alveolar epithelium. Groups of animals were then allowed to recover in 20% O2 with and without food, 40, 61, 80, and 95% O2 with food. At daily intervals, the mice were injected with tritiated thymidine to label cells preparing to divide, and sacrificed 1 hr later. Lung tissue was prepared for light microscopic autoradiography and the proportion of labeled cells were determined. In mice exposed to O3, type 1 epithelium is injured and type 2 cells proliferate in order to repair the damage. Mice allowed to recover in 20% O2 with and without food, and in 40% O2, had a maximum proliferation of type 2 cells (about eight times control) on the second day of recovery. By the fourth day repair seemed to be complete and labeling indexes had returned to normal. In mice allowed to recover in 61, 80, and 95% O2, type 2 cell proliferation was less than control on the second day and did not increase during 4 days of recovery. It was concluded that concentrations of O2 at 61% and higher inhibit reparative cell division in O3-damaged alveoli of mice." @default.
• W2036632501 created "2016-06-24" @default.
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• W2036632501 date "1981-06-01" @default.
• W2036632501 modified "2023-09-23" @default.
• W2036632501 title "Effect of high concentrations of oxygen on reparative regeneration of damaged alveolar epithelium in mice" @default.
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• W2036632501 doi "https://doi.org/10.1016/0014-4800(81)90051-4" @default.
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