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- W2037223855 abstract "TIlE ASSUMPTION TIIAT GASTROESOPllAGEAL REFLUX is the result of an incompetent lower esophageal sphincter led to the expectation that a eholinergic drug such as bethanechol, which elevates LES pressure, might be useful in its treatment, t3 Although few well-controlled clinical trials of bethanechol in GER have been performed, they have tended to support this expectation. 4-* Recent studies have shown, however, that reduced LES resting pressure is not the only factor responsible for GER. Decreased volume and buffering capacity of saliva, increased gastric emptying time, phasic relaxation of the LES, increases in intra-abdominal pressure relative to LES pressure, and inadequate esophageal clearance of acid material may all produce or perpetuate GER. 7tt On which factor(s) does bethanechol act? We have evaluated the continuous esophageal pH records of infants with GER before and immediately after initiation of bethanechol treatment, to define the early effects of bethanechol on patterns of GER and to gain information about the mode of action of this drug." @default.
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- W2037223855 date "1984-01-01" @default.
- W2037223855 modified "2023-09-26" @default.
- W2037223855 title "Bethanechol treatment of gastroesophageal reflux in infants: Effect, on continuous esophageal pH records" @default.
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- W2037223855 doi "https://doi.org/10.1016/s0022-3476(84)80609-5" @default.
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