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• W2038400855 abstract "SUMMARY Cytokines such as TNF-α and interferon gamma (IFN-γ) are important for the elimination of infected hepatocytes during acute hepatitis B virus (HBV) infection. Two G versus A transitions in the TNF-α promoter region at positions −308 and −238 possibly influence TNF-α expression. We investigated these TNF-α polymorphisms in 71 patients with chronic HBV infection, in 32 subjects that had spontaneously recovered from acute HBV infection, and in 99 healthy controls. The −238 A promoter variant was present in 18 (25%) of 71 patients with chronic HBV infection compared with two (6%) of 32 subjects with acute infection (P &lt; 0.04), and seven (7%) of 99 controls (P &lt; 0.003). By contrast, the prevalence of the variant at position −308 was similar in all investigated groups. The observed differences could not be explained by linkage disequilibrium to HLA-B or -DRB1* alleles. These findings suggest an association between the TNF-α promoter polymorphism at position −238 and the development of chronic HBV infection. This promoter variant appears to be linked to defective viral clearance." @default.
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• W2038400855 date "1998-03-01" @default.
• W2038400855 modified "2023-10-17" @default.
• W2038400855 title "A tumour necrosis factor-alpha (TNF-α) promoter polymorphism is associated with chronic hepatitis B infection" @default.
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• W2038400855 doi "https://doi.org/10.1046/j.1365-2249.1998.00534.x" @default.
• W2038400855 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/1904884" @default.
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