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• W2038764718 abstract "To elucidate the mechanisms underlying the EAE-associated behavioral syndrome (EBS), we examined the temporal correlation between the behavioral alterations and inflammatory processes. Onset of the behavioral syndrome was associated with the onset of brain infiltration, as well as mRNA expression of interleukin 1β (IL-1β) and tumor necrosis factor α (TNF-α) and elevated production of interleukin 1β protein and prostaglandin E2 (PGE2). Sickness behavior symptoms coincided with peak cytokine expression. Behavioral recovery was associated with a reduction of cytokine expression, but not infiltration, PGE2 production or motor disturbances. These results suggest that inflammatory processes in general, and the production of pro-inflammatory cytokines in particular, are involved in mediating EAE-associated sickness behavior." @default.
• W2038764718 created "2016-06-24" @default.
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• W2038764718 date "1998-11-01" @default.
• W2038764718 modified "2023-09-26" @default.
• W2038764718 title "Chemokine regulation of CNS T-cell infiltration in experimental autoimmune encephalomyelitis" @default.
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• W2038764718 doi "https://doi.org/10.1016/s0923-2494(99)80006-6" @default.
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