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• W2038864295 startingPage "2110" @default.
• W2038864295 abstract "F52 is a myristoylated, alanine-rich substrate for protein kinase C. We have generated F52-deficient mice by the gene targeting technique. These mutant mice manifest severe neural tube defects that are not associated with other complex malformations, a phenotype reminiscent of common human neural tube defects. The neural tube defects observed include both exencephaly and spina bifida, and the phenotype exhibits partial penetrance with about 60% of homozygous embryos developing neural tube defects. Exencephaly is the prominent type of defect and leads to high prenatal lethality. Neural tube defects are observed in a smaller percentage of heterozygous embryos (about 10%). Abnormal brain development and tail formation occur in homozygous mutants and are likely to be secondary to the neural tube defects. Disruption of F52 in mice therefore identifies a gene whose mutation results in isolated neural tube defects and may provide an animal model for common human neural tube defects." @default.
• W2038864295 created "2016-06-24" @default.
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• W2038864295 date "1996-03-05" @default.
• W2038864295 modified "2023-10-15" @default.
• W2038864295 title "Neural tube defects and abnormal brain development in F52-deficient mice." @default.
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• W2038864295 doi "https://doi.org/10.1073/pnas.93.5.2110" @default.
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