Matches in SemOpenAlex for { <https://semopenalex.org/work/W2039031443> ?p ?o ?g. }
- W2039031443 endingPage "112" @default.
- W2039031443 startingPage "105" @default.
- W2039031443 abstract "Excessive glutamate-mediated synaptic transmission and secondary excitotoxicity have been proposed as key determinants of neurodegeneration in many neurological diseases. Soluble mediators of inflammation have recently gained attention owing to their ability to enhance glutamate transmission and affect synaptic sensitivity to neurotransmitters. In the complex crosstalk between soluble immunoactive molecules and synapses, the endocannabinoid system (ECS) plays a central role, exerting an indirect neuroprotective action by inhibiting cytokine-dependent synaptic alterations, and a direct neuroprotective effect by limiting glutamate transmission and excitotoxic damage. On the other hand, the endocannabinoid (eCB)-mediated control of synaptic transmission is altered by proinflammatory cytokines with consequent effects in central nervous system (CNS) disorders. In this review, we summarize the interactions, at the pre- and postsynaptic level, between major inflammatory cytokines and the ECS. In addition, the behavioral and clinical consequences of the modulation of synaptic transmission during neuroinflammation are discussed. This article is part of a Special Issue entitled ‘Neuroimmunology and Synaptic Function’." @default.
- W2039031443 created "2016-06-24" @default.
- W2039031443 creator A5001899447 @default.
- W2039031443 creator A5012259287 @default.
- W2039031443 creator A5031150849 @default.
- W2039031443 creator A5079049956 @default.
- W2039031443 date "2015-09-01" @default.
- W2039031443 modified "2023-10-17" @default.
- W2039031443 title "The interplay between inflammatory cytokines and the endocannabinoid system in the regulation of synaptic transmission" @default.
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