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• W2039465863 abstract "We investigated the effect changes in end-expiratory lung volume (EEVl) had on the response to progressive hypercapnia (CO2–response curve) in eight open-chest, anesthetized dogs, in order to clarify the role that vagal lung mechanoreceptors have in altered respiratory drive during permissive hypercapnia. The dogs were ventilated using a positive-pressure ventilator driven by phrenic neural activity. Systemic arterial CO2 tension (PaCO2 ) was elevated by increasing the fraction of CO2 delivered to the ventilator. EEVl was altered from approximated functional residual capacity (“FRC”) to 1.5 and 0.5 “FRC” by changing positive end-expiratory pressure. Although the tidal volume (Vt)–PaCO2 and inspiratory time (Ti)–PaCO2 relationships were not affected, decreasing EEVl from 1.5 “FRC” to “FRC” and then to 0.5 “FRC” caused a significant (p < 0.01) upward shift in the CO2–response curves for minute ventilation (V˙ i) and frequency (f ), and a significant (p < 0.01) downward shift in the CO2– response curve for expiratory time (Te). We conclude that these shifts were explained by a decrease in the inhibitory activity of slowly adapting pulmonary stretch receptors (PSRs) as EEVl was lowered. In addition, increases in EEVl from 0.5 “FRC” to 1.5 “FRC” caused a significant (p < 0.05) increase in the apneic threshold, which we attribute to an inhibitory effect on central drive caused by increased PSR activity." @default.
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• W2039465863 date "1998-09-01" @default.
• W2039465863 modified "2023-09-27" @default.
• W2039465863 title "Control of Ventilation during Lung Volume Changes and Permissive Hypercapnia in Dogs" @default.
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• W2039465863 doi "https://doi.org/10.1164/ajrccm.158.3.9710054" @default.
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