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• W2039579255 abstract "We have used a RET-Ig fusion protein to disrupt signaling in the rat embryonic kidney development pathway. Treatment of embryonic kidney organ cultures with RET-Ig results in a decrease in branching of the ureteric bud and a down regulation in expression of the Wnt-11, Wnt-4, and ld genes. These data suggest that Wnt-11, Wnt-4, and ld function downstream of RET signaling in normal development. Expression of BMP-7, shh, and ptc were uneffected by RET-Ig treatment, implying that these genes are regulated independently of ret. We have also performed immunohistochemistry with a GFR alpha-1 specific polyclonal antisera to localize GFR alpha-1 protein expression in the developing kidney." @default.
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• W2039579255 date "1999-01-01" @default.
• W2039579255 modified "2023-10-17" @default.
• W2039579255 title "Perturbation of RET signaling in the embryonic kidney" @default.
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• W2039579255 doi "https://doi.org/10.1002/(sici)1520-6408(1999)24:3/4<263::aid-dvg9>3.0.co;2-d" @default.
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