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• W2040383454 abstract "Glis3 is a member of the Gli-similar subfamily. GLIS3 mutations in humans lead to neonatal diabetes, hypothyroidism, and cystic kidney disease. We generated Glis3-deficient mice by gene-targeting. The Glis3(-/-) mice had significant increases in the basal blood sugar level during the first few days after birth. The high levels of blood sugar are attributed to a decrease in the Insulin mRNA level in the pancreas that is caused by impaired islet development and the subsequent impairment of Insulin-producing cell formation. The pancreatic phenotypes indicate that the Glis3-deficient mice are a model for GLIS3 mutation and diabetes mellitus in humans." @default.
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• W2040383454 date "2009-05-28" @default.
• W2040383454 modified "2023-09-29" @default.
• W2040383454 title "A murine model of neonatal diabetes mellitus in <i>Glis3-</i> deficient mice" @default.
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• W2040383454 doi "https://doi.org/10.1016/j.febslet.2009.05.039" @default.
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