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• W2040464552 abstract "We investigated the influence of milacemide, a glycinamide derivative with putative antiepileptic activity, on the K+-activation of Na+,K+-ATPase in bulk isolated glial cells and synaptosomes of control and epileptogenic cortex of cats with a chronic freeze lesion. In the primary and secondary epileptic foci of non-treated animals, glial Na+,K+-ATPase lost its physiological K+-activation, while the synaptosomal enzyme was unchanged. These data reproduced previous work done on the kinetic measurement of the enzymic activities. In treated animals (500 mg/kg milacemide given orally for 2 weeks after the freeze lesion), the glial enzyme showed a normal K+-activation in the epileptic foci. These results confirm the existence of an abnormal glial Na+,K+-ATPase in cold-induced focal epilepsy and suggest that the antiepileptic activity of milacemide might be secondary to an activation of glial Na+,K+-ATPase, contributing to antagonize ictal transformation and seizure spread." @default.
• W2040464552 created "2016-06-24" @default.
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• W2040464552 date "1990-05-01" @default.
• W2040464552 modified "2023-09-27" @default.
• W2040464552 title "Milacemide stimulates deficient glial Na+, K+-ATPase in freezing-induced epileptogenic cortex of cats" @default.
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• W2040464552 doi "https://doi.org/10.1016/0006-8993(90)91022-9" @default.
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