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- W2040487176 abstract "We generated interleukin-5 receptor α chain (IL-5Rα)–deficient (IL-5Rα−/−) mice by gene targeting. The IL-5Rα−/− mice showed decreased numbers of B-1 cells concomitant with low serum concentrations of IgM and IgG3. They showed no IL-5-induced enhancement of B cell responses to T-independent antigens. The number of αβ T cell receptor–positive thymocytes tended to decrease in 3-week-old IL-5Rα−/− mice, returning to normal by 6 weeks of age. The IL-5Rα−/− mice produced basal levels of eosinophils, while their bone marrow cells failed to form eosinophilic colonies in response to IL-5. Impaired eosinophilopoiesis in IL-5Rα −/− mice enhanced the survival of Angiostrongylus cantonensis. These results indicate that IL-5-induced eosinophils serve as potent effector cells in the killing of Angiostrongylus cantonensis in mice." @default.
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- W2040487176 date "1996-05-01" @default.
- W2040487176 modified "2023-10-17" @default.
- W2040487176 title "Defective B-1 Cell Development and Impaired Immunity against Angiostrongylus cantonensis in IL-5Rα-Deficient Mice" @default.
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- W2040487176 doi "https://doi.org/10.1016/s1074-7613(00)80414-8" @default.
- W2040487176 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/8630733" @default.
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