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• W2040494217 abstract "The increasing availability of whole-genome sequence (WGS) data for Mycobacterium tuberculosis, the bacterium that causes tuberculosis (TB), suggests that circulating genotypes have been molded by three dominant evolutionary forces: long-term persistence within the human population, which requires a core programme of infection, disease, and transmission; selective pressure on specific genomic loci, which provides evidence of lineage-specific adaptation to host populations; and drug exposure, which has driven the rapid emergence of resistant isolates following the global implementation of anti-TB chemotherapy. Here, we provide an overview of these factors in considering the implications of genotypic diversity for disease pathogenesis, vaccine efficacy, and drug treatment." @default.
• W2040494217 created "2016-06-24" @default.
• W2040494217 creator A5031435647 @default.
• W2040494217 creator A5057584977 @default.
• W2040494217 creator A5072243737 @default.
• W2040494217 date "2015-01-01" @default.
• W2040494217 modified "2023-10-18" @default.
• W2040494217 title "Diversity and disease pathogenesis in Mycobacterium tuberculosis" @default.
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• W2040494217 doi "https://doi.org/10.1016/j.tim.2014.10.005" @default.
• W2040494217 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/25468790" @default.
• W2040494217 hasPublicationYear "2015" @default.
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