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• W2040782506 abstract "It has been demonstrated that VIP produces beneficial effects both in a murine model of rheumatoid arthritis and in human rheumatoid synovial fibroblasts through the modulation of proinflammatory mediators. Toll-like receptors (TLRs) play a key role in the immediate recognition of microbial surface components by immune cells prior to the development of adaptative microbe-specific immune responses. In this study, we demonstrate that VIP decreases lipopolysaccharide (LPS) and TNF-alpha-induced expression of TLR4 and its correlation with the production of CCL2 and CXCL8 chemokines in human synovial fibroblasts from patients with rheumatoid arthritis and osteoarthritis. Our results add a new step for the use of VIP, as a promising candidate, for the treatment of rheumatoid arthritis." @default.
• W2040782506 created "2016-06-24" @default.
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• W2040782506 date "2006-07-01" @default.
• W2040782506 modified "2023-09-30" @default.
• W2040782506 title "VIP Decreases TLR4 Expression Induced by LPS and TNF- Treatment in Human Synovial Fibroblasts" @default.
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• W2040782506 doi "https://doi.org/10.1196/annals.1317.045" @default.
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