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• W2040789600 abstract "Pacemaker channels play a major role in the generation of sinoatrial rhythmic activity. However, their expression is not confined to specialized myocardial cells, such as primary and subsidiary pacemakers. Electrophysiological and molecular data collected over the last ten years have demonstrated that f-channels are also present in non-pacemaker cardiomyocytes, and become upregulated in cardiac hypertrophy and failure. Mislocalized expression and/or overexpression of f-channels are a consequence of electrophysiological remodeling and, from a clinical point of view, may represent an arrhythmogenic mechanism in heart failure, a condition associated with a high risk for sudden cardiac death. The potential arrhythmogenic role of If and the availability of selective f-channel blockers cause If to be a suitable therapeutic target in heart disease." @default.
• W2040789600 created "2016-06-24" @default.
• W2040789600 creator A5005919863 @default.
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• W2040789600 date "2006-05-01" @default.
• W2040789600 modified "2023-10-16" @default.
• W2040789600 title "If in non-pacemaker cells: Role and pharmacological implications" @default.
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• W2040789600 doi "https://doi.org/10.1016/j.phrs.2006.03.015" @default.
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