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- W2040904080 abstract "Immune-neuronal interactions contribute to neuropathic pain. Thus, immune-competent cells such as microglia may provide targets for pain relief, as may infiltrating lymphocytes. We evaluated the nature of the lymphocyte response in the spinal cord in association with the maintenance of neuropathic allodynia. We assessed T cell contribution to pain processing by targeting these cells with Glatiramer acetate (GA) which when administered systemically reversed neuropathic allodynia, inhibited microglia response and increased IL-10 and IL-4 expressing T cells in neuropathic dorsal horns. These studies advance understanding of lymphocyte contribution to chronic pain and reveal a new mechanism of T cell intervention." @default.
- W2040904080 created "2016-06-24" @default.
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- W2040904080 date "2011-05-01" @default.
- W2040904080 modified "2023-09-30" @default.
- W2040904080 title "Glatiramer acetate attenuates neuropathic allodynia through modulation of adaptive immune cells" @default.
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- W2040904080 doi "https://doi.org/10.1016/j.jneuroim.2011.01.005" @default.
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