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• W2042201874 abstract "Activation of anti-apoptotic gene transcription by NF-κB (nuclear factor-kappa B) has been reported to be linked with a resistance of cancer cells against chemotherapy. NEMO (NF-κB essential modulator) interacts with a number of proteins and modulates the activity of NF-κB pathway. In this study, we revealed that RPAP3 (RNA polymerase II-associated protein 3) possesses an activity to bind with NEMO and to inhibit the ubiquitination of NEMO and that RPAP3 enhances doxorubicin-induced cell death in breast cancer cell line T-47D through the marked impairment of NF-κB pathway. These results indicate that RPAP3 may be a novel modulator of NF-κB pathway in apoptosis induced by anti-cancer chemotherapeutic agents." @default.
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• W2042201874 date "2011-01-01" @default.
• W2042201874 modified "2023-10-17" @default.
• W2042201874 title "RPAP3 enhances cytotoxicity of doxorubicin by impairing NF-kappa B pathway" @default.
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• W2042201874 doi "https://doi.org/10.1016/j.bbrc.2010.12.071" @default.
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