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• W2042483110 abstract "Agonists acting at the CB1 cannabinoid receptor in N1E-115 neuroblastoma cells were found to activate MAPK family members with reciprocal efficacies. Thus, HU 210 robustly increased phosphorylation of ERK1/2 whereas CP 55,940 was more effective in activating JNK. The use of selected kinase inhibitors confirmed that distinct signalling cascades were involved in these responses. This reciprocal control of MAPK activity was correlated with the observation that HU 210- and CP 55,940-mediated regulations of tyrosine hydroxylase gene expression were respectively impaired by MEK and JNK inhibitors. These data indicate that complex interactions of the CB1 receptor with intracellular signalling partners controlling MAPK activities may explain the apparent disparities in cellular responses to functional selective agonists." @default.
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• W2042483110 date "2008-10-23" @default.
• W2042483110 modified "2023-10-16" @default.
• W2042483110 title "Reciprocal influences of CB₁cannabinoid receptor agonists on ERK and JNK signalling in N1E-115 cells" @default.
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• W2042483110 doi "https://doi.org/10.1016/j.febslet.2008.10.022" @default.
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