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- W2043202073 endingPage "1597" @default.
- W2043202073 startingPage "1583" @default.
- W2043202073 abstract "Differential alteration of Toll-like receptor (TLR) expression in inflammatory bowel disease (IBD) was first described 10 years ago. Since then, studies from many groups have led to the current concept that TLRs represent key mediators of innate host defense in the intestine, involved in maintaining mucosal as well as commensal homeostasis. Recent findings in diverse murine models of colitis have helped to reveal the mechanistic importance of TLR dysfunction in IBD pathogenesis. It has become evident that environment, genetics, and host immunity form a multidimensional and highly interactive regulatory triad that controls TLR function in the intestinal mucosa. Imbalanced relationships within this triad may promote aberrant TLR signaling, critically contributing to acute and chronic intestinal inflammatory processes in IBD colitis and associated cancer." @default.
- W2043202073 created "2016-06-24" @default.
- W2043202073 creator A5011843830 @default.
- W2043202073 date "2010-09-01" @default.
- W2043202073 modified "2023-10-07" @default.
- W2043202073 title "Toll-like receptors in inflammatory bowel diseases: A decade later" @default.
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