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- W2043287738 abstract "In humans, perturbations in the developmental neuronal death leading to an excess of neurons could be associated with developmental neuropsychiatric disorders. Hu-bcl-2 transgenic mice appear to be a valuable tool to study the functional role of developmental programmed cell death. Indeed, the over-expression of the anti-apoptotic gene bcl-2 decreases developmental neuronal death and Hu-bcl-2 mice present supernumerary neurons in several brain regions. A detailed behavioral analysis of these mice revealed selective deficits. Hu-bcl-2 mice have normal vision, general activity and motor skills. Only the most complex behavior like anxiety and learning abilities are impaired in these mice." @default.
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- W2043287738 date "2002-01-01" @default.
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- W2043287738 title "To die or not to die, does it change the function? Behavior of transgenic mice reveals a role for developmental cell death" @default.
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- W2043287738 doi "https://doi.org/10.1016/s0361-9230(01)00639-6" @default.
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