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- W2043502417 abstract "<h2>Abstract</h2> As a result of the mechanical effect of the narrowed mitral orifice, circulation through the lungs is greatly modified in mitral stenosis. Hypertension in all segments of the pulmonary vascular bed—venous, capillary and arterial—develops as the consequence of the invariably present elevation of left atrial pressure. The cardiac output is reduced and relatively fixed. The flow resistance of the pulmonary arterioles is increased, sometimes markedly, causing an elevation of the pulmonary arterial pressure out of proportion to the pulmonary venous pressure and thereby accounting for a considerable portion of the increased pressure-work load of the right ventricle. The decrease in resistance after mitral commissurotomy suggests that physiologic, as well as previously described anatomic factors, are responsible for the arteriolar resistance. The hydrostatic pressure in the pulmonary capillaries is frequently equal to plasma colloid osmotic pressure and exceeds it during brief periods of exercise without development of frank pulmonary edema. This suggests that alterations in the alveolocapillary membrane tending to prevent the formation of pulmonary edema are present in mitral stenosis. Tricuspid insufficiency without physiologic stenosis is found in approximately one-third of patients with mitral stenosis." @default.
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- W2043502417 date "1952-06-01" @default.
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- W2043502417 title "Modifications of the pulmonary circulation in mitral stenosis" @default.
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- W2043502417 doi "https://doi.org/10.1016/0002-9343(52)90339-2" @default.
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