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• W2043661356 abstract "Pharmacological inhibition of excitatory neurotransmission attenuates cell death in models of global ischemia/reperfusion and hypoglycemia. The current investigations extend these observations to a model of focal ischemia. Kynurenic acid, a broad-spectrum antagonist at excitatory amino acid receptors, was used as treatment (300 mg/kg; 3 doses at 4-hour intervals) before and after focal cerebral ischemia in rats (n = 54). Preischemia but not 1 hour postischemia treatment with kynurenate attenuated infarction size (p < 0.001) and improved neurological outcome (p < 0.001) studied at 24 hours after injury. These data support the role of excitatory neurotransmission in acute neuronal injury and support pharmacological inhibition of cell excitation as a potential therapy for stroke." @default.
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• W2043661356 date "1987-12-01" @default.
• W2043661356 modified "2023-10-09" @default.
• W2043661356 title "Kynurenate inhibition of cell excitation decreases stroke size and deficits" @default.
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• W2043661356 doi "https://doi.org/10.1002/ana.410220609" @default.
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