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- W2044004018 endingPage "885" @default.
- W2044004018 startingPage "875" @default.
- W2044004018 abstract "Reactive oxygen species (ROS) are known to be primarily responsible for the impairment of cellular function under numerous abiotic and biotic stress conditions. In this paper, using non-invasive microelectrode ion flux measuring (MIFE) system, we show that the application of a hydroxyl radical (OH•)-generating Cu2+/ascorbate (Cu/a) mixture to Arabidopsis roots results in a massive, dose-dependent efflux of K+ from epidermal cells in the elongation zone. Pharmacological experiments suggest that both outward-rectifying K+ channels and non-selective cation channels (NSCCs) mediate such effluxes. Low (5 mM) concentrations of compatible solutes (glycine betaine, proline, mannitol, trehalose or myo-inositol) significantly reduces OH•-induced K+ efflux, similar to our previous reports for NaCl-induced K+ efflux. Importantly, a significant reduction in K+ efflux was found using osmolytes with no reported free radical scavenging activity, as well as those for which a role in free radical scavenging has been demonstrated. This indicates that compatible solutes must play other (regulatory) roles, in addition to free radical scavenging, in mitigating the damaging effects of oxidative stress." @default.
- W2044004018 created "2016-06-24" @default.
- W2044004018 creator A5003620885 @default.
- W2044004018 creator A5059041607 @default.
- W2044004018 date "2007-07-01" @default.
- W2044004018 modified "2023-09-25" @default.
- W2044004018 title "Compatible solutes reduce ROS-induced potassium efflux in Arabidopsis roots" @default.
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- W2044004018 doi "https://doi.org/10.1111/j.1365-3040.2007.01674.x" @default.
- W2044004018 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/17547658" @default.
- W2044004018 hasPublicationYear "2007" @default.