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- W2044480541 abstract "Several studies have consistently demonstrated that inflammation plays a key role in the pathogenesis of acute coronary syndromes (ACS) [ 1 Liuzzo G. Biasucci L.M. Gallimore J.R. et al. The prognostic value of C-reactive protein and serum amyloid a protein in severe unstable angina. N Engl J Med. 1994; 331: 417-424 Crossref PubMed Scopus (2095) Google Scholar , 2 Blake G.J. Ridker P.M. C-reactive protein and other inflammatory risk markers in acute coronary syndromes. J Am Coll Cardiol. 2003; 41: 37S-42S Abstract Full Text Full Text PDF PubMed Google Scholar ]. More recent studies have highlighted the importance of adaptive immunity in ACS [ 3 Caligiuri G. Paulsson G. Nicoletti A. Maseri A. Hansson G.K. Evidence for antigen-driven T-cell response in unstable angina. Circulation. 2000; 102: 1114-1119 Crossref PubMed Scopus (112) Google Scholar , 4 De Palma R. Del Galdo F. Abbate G. et al. Patients with acute coronary syndrome show oligoclonal T-cell recruitment within unstable plaque: evidence for a local, intracoronary immunologic mechanism. Circulation. 2006; 113: 640-646 Crossref PubMed Scopus (102) Google Scholar ]. In particular, profound abnormalities have been observed in specific subsets of T-cells, including CD4+CD28nullT-cells, a subset of cytotoxic CD4+T-lymphocytes producing large amount of interferon-γ (IFN-γ) [ 5 Liuzzo G. Kopecky S.L. Frye R.L. et al. Perturbation of the T-cell repertoire in patients with unstable angina. Circulation. 1999; 100: 2135-2139 Crossref PubMed Scopus (341) Google Scholar , 6 Liuzzo G. Biasucci L.M. Trotta G. et al. Unusual CD4+CD28null T-lymphocytes and recurrence of acute coronary events. J Am Coll Cardiol. 2007; 50: 1450-1458 Abstract Full Text Full Text PDF PubMed Scopus (184) Google Scholar , 7 Giubilato S. Liuzzo G. Brugaletta S. et al. Expansion of CD4+CD28null T-lymphocytes in diabetic patients: exploring new pathogenetic mechanisms of increased cardiovascular risk in diabetes mellitus. Eur Heart J. 2011; 32: 1214-1226 Crossref PubMed Scopus (81) Google Scholar ], naturally occurring regulatory T-cells (Treg) [ 8 Mor A. Luboshits G. Planer D. Keren G. George J. Altered status of CD4(+)CD25(+) regulatory T cells in patients with acute coronary syndromes. Eur Heart J. 2006; 27: 2530-2537 Crossref PubMed Scopus (265) Google Scholar , 9 Han S. Liu P. Zhang W. et al. The opposite-direction modulation of CD4+CD25+ Tregs and T helper 1 cells in acute coronary syndromes. Clin Immunol. 2007; 124: 90-97 Crossref PubMed Scopus (106) Google Scholar ] and interleukin (IL)-17-producing T-cells (Th17) [ 10 Cheng X. Yu X. Ding Y.J. et al. The Th17/Treg imbalance in patients with acute coronary syndrome. Clin Immunol. 2008; 127: 89-97 Crossref PubMed Scopus (393) Google Scholar , 11 Li Q. Wang Y. Chen K. et al. The role of oxidized low-density lipoprotein in breaking peripheral Th17/Treg balance in patients with acute coronary syndrome. Biochem Biophys Res Commun. 2010; 394: 836-842 Crossref PubMed Scopus (47) Google Scholar , 12 Zhao Z. Wu Y. Cheng M. et al. Activation of Th17/Th1 and Th1, but not Th17, is associated with the acute cardiac event in patients with acute coronary syndrome. Atherosclerosis. 2011; 217: 518-524 Abstract Full Text Full Text PDF PubMed Scopus (69) Google Scholar ]." @default.
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- W2044480541 title "Identification of unique adaptive immune system signature in acute coronary syndromes" @default.
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