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- W2044514664 abstract "The innate immune system consists of functionally specialized modules that are activated in response to a particular set of stimuli via sensors located on the surface or inside the tissue cells. These cells screen tissues for a wide range of exogenous and endogenous danger/damage-induced signals with the aim to reject or tolerate them and maintain tissue integrity. In this line of thinking, inflammation evolved as an adaptive tool for restoring tissue homeostasis. A number of diseases are mediated by a maladaptation of the innate immune response, perpetuating chronic inflammation and tissue damage. Here, we review recent evidence on the cross talk between innate immune sensors and development of rheumatoid arthritis, osteoarthritis, and aseptic loosening of total joint replacements. In relation to the latter topic, there is a growing body of evidence that aseptic loosening and periprosthetic osteolysis results from long-term maladaptation of periprosthetic tissues to the presence of by-products continuously released from an artificial joint." @default.
- W2044514664 created "2016-06-24" @default.
- W2044514664 creator A5037442831 @default.
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- W2044514664 creator A5053817793 @default.
- W2044514664 creator A5054831892 @default.
- W2044514664 creator A5070363000 @default.
- W2044514664 date "2014-01-01" @default.
- W2044514664 modified "2023-10-03" @default.
- W2044514664 title "Innate Immunity Sensors Participating in Pathophysiology of Joint Diseases: A Brief Overview" @default.
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