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• W2044681902 abstract "Nitric oxide (NO) signaling is required for the induction of long-term depression (LTD) in parallel fiber/Purkinje cell synapses in the cerebellum: NO is released following climbing fiber stimulation and blockade of this NO signal suppresses LTD in slices. However, NO is not obligatory for inducing LTD in cultured Purkinje cells or in slices treated with fluorocitrate, a gliotoxic metabolic inhibitor. Various possibilities about the roles of NO in cerebellar synaptic plasticity are discussed: NO may enable LTD by removing some inherent glial suppression on neural plasticity; it may work as an integrated signal producing some decision-making ability to cerebellar neural networks; and it may coordinate quick adaptation and life-time stability in cerebellar motor learning." @default.
• W2044681902 created "2016-06-24" @default.
• W2044681902 creator A5024840673 @default.
• W2044681902 date "1993-06-01" @default.
• W2044681902 modified "2023-09-25" @default.
• W2044681902 title "Nitric oxide: a multi-functional messenger substance in cerebellar synaptic plasticity" @default.
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• W2044681902 doi "https://doi.org/10.1016/s1044-5765(05)80055-7" @default.
• W2044681902 hasPublicationYear "1993" @default.
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