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• W2044687413 abstract "<i>Introduction:</i> The aim of this study was to assess the expression and production of inflammation mediators in basal condition and after angiotensin II (AngII) in otosclerosis. <i>Materials and Methods:</i> Human stapedial cell cultures (6 otosclerosis and 6 controls) were incubated with AngII (10^–7<i>M</i>, 24 h) or vehicle. Cytokines and their mRNA expression were assessed by antibody and cDNA arrays. <i>Results:</i> In basal conditions, otosclerotic cultures produced higher amounts of interleukin (IL)-1β and interferon-inducible protein 10, and smaller amounts of tissue inhibitor of metalloproteinase 2. AngII promoted inflammation by increasing interferon γ and IL-10, and by decreasing macrophage inflammatory protein 1α and soluble tumor necrosis factor receptor II. <i>Conclusions:</i> Otosclerotic cultures produced higher proinflammatory cytokines in basal condition. AngII appeared to promote inflammation via these mediators in otosclerosis." @default.
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• W2044687413 date "2012-01-01" @default.
• W2044687413 modified "2023-10-17" @default.
• W2044687413 title "Effect of Angiotensin II on Inflammation Pathways in Human Primary Bone Cell Cultures in Otosclerosis" @default.
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• W2044687413 doi "https://doi.org/10.1159/000335098" @default.
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